Browsing by Author "A.N. Rai"
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PublicationArticle Carbohydrate metabolism in cirrhosis liver in tropics(1979) J.P. Gupta; H.S. Bajpai; O.P. Rai; A.N. RaiThe abnormalities of carbohydrate metabolism in hepatic disorders have been reported by various workers from the continent, however, relatively few reports are available from India where the syndrome of chronic hepatic failure, cirrhosis of the liver, differs from that of the West. A project has been undertaken to study the carbohydrate abnormalities in hepatic disorders with special reference to cirrhosis of the liver along with observations on insulin, a key hormone in carbohydrate metabolism.PublicationBook Cyanobacteria: From Basic Science to Applications(Elsevier, 2018) A.K. Mishra; D.N. Tiwari; A.N. RaiCyanobacteria constitute the most widely distributed group of photosynthetic prokaryotes found in almost all realms of the earth and play an important role in Earth's nitrogen and carbon cycle. The gradual transformation from reducing atmosphere to oxidizing atmosphere was a turning point in the evolutionary history of the earth and made conditions for present life forms possible. Cyanobacteria: From Basic Science to Applications is the first reference volume that comprehensively discusses all aspects of cyanobacteria, including the diverse mechanisms of cyanobacteria for the advancement of cyanobacterial abilities, towards higher biofuel productivity, enhanced tolerance to environmental stress and bioactive compounds and potential for biofertilizers. © 2019 Elsevier Inc. All rights reserved.PublicationArticle Effect of cyanophage N-1 development on nitrogen metabolism of cyanobacterium Nostoc muscorum(1988) A.K. Kashyap; A.N. Rai; Surendra SinghThe impact of cyanophage N-1 development on nitrogenase, glutamine synthetase (GS) and aminotransferases activities in the diazotrophic cyanobacterium Nostoc muscorum was investigated during its latent period. The nitrogenase activity was inhibited after 2 h of infection, suggesting that phage development does not require the product of nitrogenase activity. GS activity was not inhibited until 4 h of infection; however, a decline in activity was subsequently observed. Glutamate oxaloacetate transaminase was inhibited after 1 h of infection and no activity was detectable during the entire latent period. In contrast, glutamate pyruvate transaminase activity increased 2-fold by 4 h of infection and remained higher than the background level until the end of the latent period. The results suggested that under nitrogen fixing conditions, N-1 multiplication proceeds in the absence of nitrogen fixation and that the metabolism of amino acids is altered in favour of phage multiplication. © 1988.PublicationArticle Estimation of average life for negative exponential distribution(1993) Umesh Singh; S.K. Upadhyay; A.N. RaiThis paper deals with the problem of average life estimation when the life times follow negative exponential distribution. A minimum mean square error estimator belonging to the class of linear combination of complete sufficient statistics has been obtained. The estimator thus obtained requires knowledge of coefficient of variation. The effect of using a guessed value of coefficient of variation on the estimator has also been studied. © 1993.PublicationEditorial PublicationEditorial PublicationArticle Pulmonary embolism in rheumatic heart disease(1973) A.N. Rai; S.S. Singh; A.P. PandeyPulmonary embolism is a known complication of phlebothrombosis affecting venous system of legs, pelvis and abdomen. Smith et al. (1965) reported that the commonest site of thrombus formation is leg veins (46%), followed by right atrium (38%), inferior vena cava (19%), pelvic veins (16%) and right ventricle (4%), in the order of frequency. Though pulmonary embolism may occur in apparently healthy individuals (Homan, 1943 and Short, 1952), heart disease patients are more at risk (Carlotti et al., 1947, Short, 1952, Byrne, 1960, Sasahara et al., 1967). Sasahara et al. (1967) found that 69% of their cases of pulmonary embolism had cardiac disease, 21% of whom had rheumatic mitral disease. In an earlier study by Sasahara (1965), 50% of the cases of pulmonary embolism were found to have rheumatic heart disease. The present work was undertaken in order to study different aspects of pulmonary embolism in rheumatic heart disease.PublicationArticle Radioimmunoassay of serum digoxin in relation to digoxin intoxication(1975) R.B. Singh; A.N. Rai; K.P. Dube; D.K. Srivastav; P.N. Somani; B.C. KatiyarSerum digoxin estimations were done in 98 patients receiving digoxin for heart failure of varied aetiology. Digoxin toxicity or the lack of it was determined on the basis of established electrocardiographic criteria. Fifty two patients were classified as 'toxic' and 46 as 'non toxic'. The difference in the mean digoxin levels between the two groups was highly significant (P<0.001). The mean serum digoxin level in 'non toxic' patients was slightly higher than that found by other investigators. Fairly good correlations have been noted between different dosage schedules and various rhythm disturbances. Death was attributed to digoxin toxicity in only 2 patients who showed electrocardiographic evidence of intoxication at the time of death.PublicationArticle Risk factors of digoxin intoxication(1975) R.B. Singh; S.K. Vaish; A.N. Rai; K.P. Dube[No abstract available]PublicationArticle Situs inversus with amoebic liver abscess(1973) Z.A. Ansari; J. Skaria; M.S. Gopal; S.K. Vaish; A.N. Rai[No abstract available]