Browsing by Author "B.V. Agrawal"
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PublicationArticle Aetiology of hypertension in young age(1974) S.K. Vaish; B.V. Agrawal; A.K. Srivastava; V. Prasad; K.P. Dubey; P.K. Das[No abstract available]PublicationArticle Amebic pericardial effusion: a rare complication of amebic liver abscess(1975) B.V. Agrawal; P.N. Somani; M.N. Khanna; P.K. Srivastava; B.N. Jha; S.P. VermaTwo rare cases of amebic pericardial effusion as a complication of amoebic liver abscess in the left lobe are described. The pericardial amebiasis should be suspected in a patient presenting with signs and symptoms of pericardial effusion with an evidence of hepatic abscess (in the left lobe) or in a patient with pericardial effusion of uncertain etiology. Aspiration of ''anchovy sauce'' pus from both the pericardial cavity and the liver should be regarded as confirming the diagnosis of amebic pericarditis secondary to amebic liver abscess because demonstration of Entamoeba histolytica is seldom possible. Removal of pericardial pus and metronidazole intake were markedly effective in treating our patients.PublicationArticle Cardiac asystole due to lignocaine in a patient with digitalis toxicity(1974) B.V. Agrawal; R.B. Singh; S.K. Vaish; H. EdinAn elderly female who had unusual manifestations of digitalis sensitivity and toxicity is presented. During treatment of digitoxic ventricular tachycardia by a 50 mg bolus of lignocaine both ventricular and atrial asystole (a previously undescribed manifestation of lignocaine sensitivity or toxicity) developed in this arrhythmia is discussed. Lignocaine in patients with ventricular dysrhythmias due to digitalis overdosage should be used with caution or better avoided.PublicationArticle Clinical trial of nifedipine in angina pectoris(1988) P.N. Somani; B.V. Agrawal; A.G. Rajan[No abstract available]PublicationArticle Corrected QT interval (QTc) and its relationship with fatal ventricular tachyarrhythmias and sudden death in acute myocardial infarction(1984) P.R. Gupta; A. Kumar; V.P. Singh; B.V. Agrawal; P. Awasthey; P.N. Somani[No abstract available]PublicationArticle Dextrocardia with myocardial infarction(Indian Medical Association, 1995) S. Chakravorty; P.C. Sarma; B.V. Agrawal[No abstract available]PublicationLetter PublicationArticle Left bundle branch block: a rare manifestation of digitalis intoxication(1976) R.B. Singh; B.V. Agrawal; P.N. Somani[No abstract available]PublicationArticle Left ventricular function in acute myocardial infarction before and after oxyfedrine(1985) B.V. Agrawal; V.K. Agrawal; P.N. Somani[No abstract available]PublicationConference Paper Left ventricular mass regression with amlodipine in elderly hypertensives(Marcel Dekker Inc., 1999) N.K. Singh; S.K. Gupta; B.V. AgrawalWe evaluated effect of amlodipine therapy on left ventricular (LV) mass and function indices in 34 elderly hypertensives having echocardiographic evidence of left ventricular hypertrophy (LVH). LV mass and LV function indices were evaluated before and after 4 months of effective amlodipine therapy. Fourteen patients completed the study. Blood pressure was effectively controlled in all the patients; dose requirement of amlodipine being 5 mg/day in 8 and 10 mg/day in 6 patients. We found significant regression of LV mass indices such as thickness of interventricular septum (IVS) and left ventricular posterior wall (LVPW), and LV mass index after 4 months of amlodipine therapy. However, LV function indices did not alter following treatment. In conclusion, amlodipine therapy besides effectively controlling BP in elderly hypertensives, produced significant regression of LV mass indices without affecting the LV function.PublicationArticle Observations on atrial tachycardia with block(1978) B.V. Agrawal; P.N. Somani; P.K. Jain; P.K. Srivastava[No abstract available]PublicationArticle PublicationArticle QTc interval in pulmonary tuberculosis(1982) P.K. Jain; B.V. Agrawal; P. Avasthey; P.N. Somani; V.K. JhaThe mean QTc interval in 140 normal adults was 0.40 second (S.D. ± 0.02, range 0.32 to 0.44 second). It was 0.44 second (S.D. ± 0.09, range 0.32 to 0.54 second) in 125 patients with pulmonary tuberculosis. The difference was statistically highly significant (p<0.001). The QTc interval was prolonged (more than 0.44 second) in 47.2% of cases of pulmonary tuberculosis. It was not affected by the duration or extent of pulmonary lesion, elevated body temperature or sedimentation rate. The increase in QTc interval appears to be due to myocardial involvement of tuberculosis.PublicationArticle Serum immunoglobulins in rheumatic heart disease(1975) B.V. Agrawal; R.M. Gupta; V.P. Singh; P.N. SomaniSerum IgG, IgA and IgM all were elevated in rheumatic heart patients. IgG and IgA had statistically significant raised values. Serum IgM was much more increased in acute rheumatic heart disease while in chronic rheumatic heart patients IgA was more elevated. Patients with congestive cardiac failure had more increased levels of serum IgA (p < 0.001). Patients belonging to class III and IV of functional cardiac status (virtually decompensated) had raised serum IgA levels. There was no significant difference in Ig levels in patients with rheumatic heart disease with and without associated infections. The duration of heart disease did not alter Ig levels significantly. The alteration in serum immunoglobulins of patients with rheumatic heart disease thus suggests an immunological disturbance related to the disease process.PublicationArticle Staphylococcal myocarditis presenting with Stokes-Adams attacks(1981) B.V. Agrawal; S.P. Verma; A. Sharma; P.N. Somani; P.K. Srivastava[No abstract available]PublicationArticle Twin immunologic test (TIT) in myocardial infarction. A reliable laboratory index(1989) R.M. Gupta; P. Awasthey; B.V. Agrawal; P.R. Gupta; S. Usha; V. Ja iswal[No abstract available]