Publication:
Birth hypoxia and spinal reflex in newborn babies

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dc.contributor.authorPrakash, Udai
dc.contributor.authorSinha, B.
dc.contributor.authorBhatia, B.D.
dc.date.accessioned2025-03-04T05:56:36Z
dc.date.available2025-03-04T05:56:36Z
dc.date.issued2005
dc.description.abstractBirth hypoxia is detrimental to neuronal function. In this study, its effect on spinal monosynaptic reflex was investigated on two different age groups of human newborn babies using few non-invasive electrophysiological parameters. A total of 57 newborns (25 hypoxic + 32 non-hypoxic) were the study subject. Out of which, 31 newborns (11 hypoxic + 20 non-hypoxic) were examined within 48 hours of birth, and the rest 26 (14 hypoxic + 12 non-hypoxic) were examined between 48h and 120h (five days) of birth. H-reflex latency (HRL), distal motor latency (DL), and H-reflex conduction velocity (HRCV), were estimated for understanding the transmission of impulse in the monosynaptic reflex pathway. Moreover, Hmax, Mmax, and HIM ratio were observed for studying the spinal motor neuronal excitability. The injurious effect of hypoxia was detected on HRL, HRCV, Hmax and HIM ratio in babies who were examined within 48h of birth. The HRL was significantly increased and other parameters were significantly reduced. In the older group of babies, however, Hmax was the only parameter affected by hypoxia. The relatively older babies of hypoxic or non-hypoxic group had higher Hmax and HIM ratio compared to younger ones of their own group.
dc.identifier.doihttps://doi.org/nan
dc.identifier.issn0301150X
dc.identifier.urihttps://dl.bhu.ac.in/ir/handle/123456789/87726
dc.subjectBirth hypoxia
dc.subjectH-reflex
dc.subjectNeuronal excitability
dc.subjectNewborns
dc.subjectReflex latency
dc.titleBirth hypoxia and spinal reflex in newborn babies
dc.typeArticle
dspace.entity.typePublication
journal.titleElectromyography and Clinical Neurophysiology
journalvolume.identifier.volume45

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2005